Obesity is one of the most dangerous of problems confronting America today. A recent study showed that an incredible 65% of American adults are either overweight or obese Ã¢â‚¬â€œ a ridiculously staggering amount that is unequaled in the entire world. The same study reported that 31% of all American children are classified as overweight or obese.
As Americans eat more food, it seems as though they are exercising less and less.
An estimated 40% of adults and teenagers do not exercise on a regular basis. With increasing rates of food consumption and decreasing rates of physical activity comes higher rates of illness. With the health of the average American deteriorating, the nation has been beset with a host of diseases like diabetes, heart failure, and yes, prostate cancer.
Does good nutrition really make a difference on your risk of developing prostate cancer? The old saying Ã¢â‚¬Å“you are what you eatÃ¢â‚¬Â may well sum up the answer! There are some risk factors you cannot change. Age, family history, and race are all-important components to consider when assessing risk factors for cancer. One or all can play an important role on your chances of developing cancer.
Age is the leading factor in your risk in developing prostate cancer. The older you are the more likely you re to having prostate problems. Under the age of forty you have a one in ten thousand chance of developing this disease. After the age of 40 your risk rate goes up dramatically.
Statistics say that 90 percent of men over the age of 70 will develop prostate problems or prostate cancer. African-American men have a higher risk of developing prostate cancer and those men who have a close male relative with the disease are at increased risk.
Other Risk Factors
Although age is a risk factor for prostate cancer there are other things that can influence your risk. Your diet and lifestyle can reduce or increase your risk of developing prostate cancer. The body has natural defenses built in to stop good cells from turning into cancer and to slow the growth of cells that are cancerous.
The diagnosis of HodgkinÃ¢â‚¬â„¢s Lymphoma (HL) usually depends on people having abnormal cells, called Reed-Sternberg (R-S) cells, in their blood. Other types of abnormal cell types may be present as well.
However, the presence of R-S cells alone does not necessarily mean that an individual definitely has HodgkinÃ¢â‚¬â„¢s Lymphoma.
To confirm a diagnosis, the lymphatic tissue that contains Reed-Sternberg cells must also be surrounded by a background of other cells and features that are characteristic of HodgkinÃ¢â‚¬â„¢s Lymphoma. A pathologist may use immunological tests that look for cell surface markers (antigens) that identify specific cell types in order to help confirm or otherwise a diagnosis for HodgkinÃ¢â‚¬â„¢s Lymphoma.
HL has been studied more than any other type of Lymphoma. With the many rapid advances in diagnosis and treatment, over 80% of patients with HL can be cured.
Nonsmall cell lung cancer is more widespread than small cell lung cancer, and generally it grows and spreads more gradually. The remaining lung cancers are all non-small cell (NSCLC).
There are three sub-types of NSCLC. The cells in these sub-types fluctuate in size, shape, and chemical make-up.
Squamous cell carcinoma: About 25% – 30% of all lung cancers are Squamous cell carcinomas. They are linked with a history of smoking and tend to be found centrally, near a bronchus.
Squamous cell carcinoma generally starts in the bronchi and doesn’t usually spread as rapidly as do other lung cancers
Adenocarcinoma: This type of cancer accounts for about 40% of lung cancers and is typically found in the outer region of the lung. Adenocarcinoma is more commonly found in women than in men.
Large-cell undifferentiated carcinoma: This kind of cancer accounts for about 10% – 15% of reported lung cancers. It appears in any part of the lung and tends to grow and spread quickly throughout the body ensuing in a poor prediction of recovery.
Large cell carcinoma is any lung tumor that cannot be classified
As cigarette smoking is a major cause of lung cancer nowadays, it is important to appreciate how smoking affects and injures the lungs. This is because smoke inhalation damages the normal cleaning process by which the lung protects itself from injury.
The bronchi which conduct inhaled air to the lung tissues are lined with a single coating of cells on which lies a defensive coating of mucus. The hair-like cilia on these cells beat in a regular rhythm to advance mucus upwards continually from the lung removing any inhaled particles which may have become trapped in the process.
The competence of this cleaning mechanism is damaged very quickly by smoke inhalation. The cilia disappear and the coating they lie in thickens in an attempt to protect the fine underlying tissues from injury. Once this damage has occurred, the lung can no longer keep itself uncontaminated.
As a result, the cancer-producing agents in cigarette smoke remain ensnared in the mucus on the surface lining of the airway. They then pass into the cells before being removed by coughing which is the only cleansing mechanism remaining.
Once they are within the body, these chemicals, and their by-products, alter the very nature of the cells in the lungs slowly and increasingly until finally cancer develops.